In the wake of a deadly, widespread outbreak of coronavirus in the US, scientists have begun to explore the role that genes play in the spread of the virus.
Scientists are currently studying a gene called “paranorm activity” that has been found in mice with a mutation that leads to a stronger response to viruses like coronaviruses.
Paranorm activation, as the study is now called, allows people to live longer, healthier lives, but also makes it more likely that those who survive the virus will be able to spread it to other people, and more importantly, to those they come into contact with.
The gene has been linked to a genetic variant called COVID-19 that is found in a gene in humans called CYP2D6.
That gene, like other genes, controls whether a person is susceptible to a particular coronaviral strain or not.
It is a gene that has also been linked in other studies to obesity, diabetes and cancer.
The new study, published in the journal PLOS ONE, looked at a single mutation in a specific gene called COV-2S1.
“Paranormal Activity” occurs when an individual has a mutation in their CYP1D1 gene that makes it resistant to COVID.
This mutation causes those who have it to develop a higher rate of COVID activity.
People who have this mutation have the same risk of contracting COVID as those who do not.
The researchers then took this mutation and looked at the effects on human cells in response to COV, using a technique called immunohistochemistry.
Their findings show that the mutation leads to an increase in the levels of the enzyme activator protein (IAP), which is produced by cells that respond to COVR-1.
This protein is responsible for the production of antibodies against COVRs, which in turn increases the likelihood that an infected person will be contagious.
IAP is also associated with an increased rate of infections in people with weakened immune systems, and this finding suggests that people who have low levels of IAP may be more vulnerable to COVEV-1 than those with normal levels.
“The findings are exciting and important for understanding the mechanisms of COVEv1 transmission and the potential for COV vaccines to be effective against the virus,” said lead author Yuki Takahashi, an immunologist at the University of California, Berkeley.
“[They] also provide a useful biomarker for understanding COVEs susceptibility to treatment.”
It is unclear how long the mutation will persist in humans, but scientists are hoping that we will have a better understanding of the link between it and longevity and cancer as a result.
If this gene mutation is found to be in humans with a COVID mutation, it could be the first time that a gene has caused a significant increase in mortality in the human population, and could also help scientists understand how the virus works.
Read more about coronavivirus here: https://www.reddit.com/r/science/comments/4gjxqp/the_world_is_on_the_defining_edge_of_a/